Selenium deficiency in poultry biology essay
It is well known that dietary supplementation of selenium, vitamin E and carotenoids can modulate antioxidant defense in poultry. The aim of this article is to present evidence regarding the modulation of antioxidant capacities in poultry by vitamin E. model systems, including male poultry farmers, semen, and selenium deficiency is known to cause cardiovascular disease. However, the role of Se deficiency in causing oxidative damage and inflammatory injury to chicken aortic vessels is not well understood. In the present study, day-old chicks were randomly divided into two groups, a low Se group L and a control Se group C. Previous studies have investigated the effects of dietary selenium Se supplementation on selenoprotein N SelN, SEPN1 , certain. selenophosphate synthetase-1 SPS1 and selenocysteine synthase SecS mRNA abundance in chicken skeletal and cardiac muscle. To investigate the collective responses of these genes to dietary Se, the biological trace element selenium Se is crucial for various life forms. Se deficiency can impair immune function and cause DNA damage in chickens. Studies have shown that Se deficiency negatively affects immune cells during activation, differentiation, and proliferation, causing protein and calcium misfolding. Recently emerged viral infectious diseases VIDs include HIV AIDS, influenza H5N1 H1N1, SARS and Ebola hemorrhagic fever. Previous research has identified metabolic oxidative stress in hosts deficient in antioxidant selenium Se. 1 µMol Se L blood induces reduced immunocompetence of the human host as well as rapidly. two ER-resident selenoproteins, namely Selenok and Selenom, in the ovaries of aging mice. The mRNA expression of Selenok and Selenom RT-qPCR was significantly higher in the ovaries of mice fed diets, Antioxidant systems in poultry biology: nutritional modulation of vitagenes Systeme in der Gefl gelphysiologie: 2012: Effects of oxidative stress on immunosuppression induced by selenium deficiency in chickens . Biol. Trace Elem. Res. 149, 352-361. Zhao. We first wondered whether dietary selenium deficiency regulated apoptosis in chicken liver. The treated diet was added to the daily diet. Apoptosis, one mode of cell death, was detected under TUNEL staining and TEM Fig. 1A and B. On TUNEL staining of the d-treated chicken group, the Se deficiency group showed a brighter spot than the normal diet. Feed Se level can regulate SelN biosynthesis by affecting the levels of SPS SecS RNA and regulating the expression of SelN gene and affecting the mRNA levels of SecS and S PS1. Previous studies have determined the effects of dietary selenium Se supplementation on selenoprotein N SelN, SEPN1, selenophosphate, 1. Introduction. Selenium Se is an essential trace element required for a variety of biological functions in animals, including cardiovascular function. Se deficiency has been associated with several cardiovascular diseases, including cardiomyopathies such as Keshan KD disease, 1, 2 heart failure, 3, 4 and myocardial infarction. 5 We first wondered whether dietary selenium deficiency regulated apoptosis in chicken liver treated diet added to daily diet. Apoptosis, one mode of cell death, was detected under TUNEL staining and TEM. Fig. and B. Upon TUNEL staining of the d-treated chicken group, the Se deficiency group showed a brighter spot than the normal diet. Selenium Se is an essential micronutrient present in human diet, comprising the composition of selenoproteins as selenocysteine Se-Cys amino acid. At the thyroid level.,